Exercise and Caffeine Prevent Skin Cancer

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The risk of developing skin cancers caused by exposure to the sun may be cut down by exercise and the intake of caffeine. Fat and tumor growth are linked, and that exercise combined with caffeine may prevent not only skin cancer but also inflammation linked to other cancers that are related to obesity. Most skin cancers are caused by long term exposure to the sun. There are two types of non melanoma skin cancer, basal cell carcinoma (BCC) and squamous cell carcinoma (SCC). A history of sunburn increases the risk of BCC. Risk is especially high if you had several episodes of sunburn in childhood.

The risk of SCC is linked to overall sun exposure through your life. This means that outdoor workers have an increased risk, such as, farm workers, gardeners and building site workers. Sunburn in childhood is also linked to SCC in some studies. Fair skinned people, with light colored hair and eyes, or those more likely to burn than tan, are more at risk of sun damage than dark skinned people. You have a higher risk of skin cancer if you are older or have had skin cancer before. Having a family history of skin cancer also increases risk. Other factors include having had certain other skin conditions or previous radiotherapy, having been exposed to certain chemicals and having a weakened immune system.Caffeine consumption coupled with some exercises may ward off skin cancer.

Whereas, exercise is the physical exertion of the body – making the body do a physical activity which results in a healthy or healthier level of physical fitness and both physical and mental health, the caffeine acts selectively, causing the abnormal cells to die but not affecting the normal cells. This is not a sun screening effect. It is a biological effect. There is a possibility that you can cut your skin cancer risk by 50-70% – no matter what chemicals you choose to rub on your skin or how much sun exposure you have received. Administration of caffeine enhances the removal of DNA-damaged cells by inhibiting the ATR-mediated phosphorylation of Chk1 and prematurely increasing the number of cyclin B1-containing cells that undergo lethal mitosis. Administration of green tea, coffee and caffeine may inhibit UVB-induced carcinogenesis at least in part, by enhancing UVB-induced apoptosis. Plasma levels of caffeine observed after its oral administration at cancer-preventive dose levels were within the range observed in moderate coffee drinkers.